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The Ultimate Health Podcast
561: The Top Foods You Need to Eat & Avoid to Fix Your Mitochondria & Metabolic Health | Dr. Rob Lustig
Sep 28, 2023
Neuroendocrinologist Dr. Robert Lustig discusses the root cause of insulin resistance, types of fats, recommended supplements, the impact of glucose and fructose on mitochondrial function, and the connection between insulin resistance and mitochondrial dysfunction. Various methods to measure sugar consumption and its impact on metabolic health are explored, along with the effectiveness of supplements for mitochondrial function. The podcast also delves into the history of processed food and sugar, strategies for quitting sugar addiction, myths about milk consumption, the pros and cons of calcium supplements, nutrient deficiencies in a plant-based diet, the dangers of food processing, and the impact of sweeteners on health.
02:50:34
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Quick takeaways
- Mitochondrial dysfunction is the root cause of insulin resistance and chronic metabolic diseases.
- Supplementing with omega-3 fatty acids and mono-unsaturated fats can improve mitochondrial function and metabolic health.
Deep dives
Insulin resistance: The downstream effect of mitochondrial dysfunction
Insulin resistance occurs when the mitochondria, the powerhouse of cells, are not functioning properly. Mitochondrial dysfunction is the root cause of various chronic metabolic diseases. Insulin, the energy storage hormone, helps regulate glucose and fat levels in the body. When the mitochondria are not working efficiently, insulin resistance occurs, leading to higher blood glucose and fat levels. One major contributor to mitochondrial dysfunction and insulin resistance is the accumulation of fat in the liver. Sugar, especially fructose, plays a significant role in the development of fatty liver and subsequent insulin resistance. Other factors, such as poor diet, exposure to obesogens and toxins, and radiation, can also contribute to mitochondrial dysfunction. It is crucial to address these factors to improve metabolic health and prevent or reverse insulin resistance.
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