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High Intensity Health with Mike Mutzel, MS
Fructose & Sugar Make Fat Loss Impossible: New Research
Mar 14, 2024
New research suggests that fructose and sugar are major drivers of obesity and metabolic disease, impacting energy levels, leptin signaling, and aging pathways. High carb and salt diets increase diabetes risk, while Berberine fasting accelerator may support intermittent fasting. Excessive fructose metabolism can disrupt metabolic pathways, increase body fat storage, and promote hyperphagia. Uric acid and liver enzyme elevation may serve as biomarkers for health complications.
13:20
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Quick takeaways
- Fructose survival hypothesis explains obesity as an ancient survival switch overstimulated by modern hyper-palatable junk food.
- Excessive fructose consumption can lead to metabolic issues like insulin resistance, inflammation, and weight gain.
Deep dives
Fructose Survival Hypothesis and Evolutionary Context
The podcast discusses the fructose survival hypothesis, proposing an evolutionarily adaptive mechanism exploited in modern society with easy access to hyper-palatable, ultra-processed junk food. This hypothesis suggests that obesity and metabolic disorders stem from an overstimulation of an ancient survival switch aimed to protect animals in times of crisis, like food shortage. The phenomenon leads to obesity, metabolic diseases like diabetes and dementia, high blood pressure, cardiovascular issues, and accelerated aging.
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Discover
highlights
Listen to the best highlights from the podcasts you love and dive into the full episode