There's exactly the same number of nerons in every c elegance. And for aging, that has some serious implications. For example, if you think about it, like there's this insolent receptor mutin called daftoo, which lives twice as long as wild type. It doesn't do that by turning over rid of damaged cells and growing new ones. It has to do it by keeping those cells healthy the whole life span. So what should you do to keep cell that you may have your whole life, like a brain cell? Am, what would you have to do to keep that cell alive?
Aging -- everybody does it, very few people actually do something about it. Coleen Murphy is an exception. In her laboratory at Princeton, she and her team study aging in the famous C. Elegans roundworm, with an eye to extending its lifespan as well as figuring out exactly what processes take place when we age. In this episode we contemplate what scientists have learned about aging, and the prospects for ameliorating its effects -- or curing it altogether? -- even in human beings. Coleen Murphy received her Ph.D. in biochemistry from Stanford University, and is currently Professor in the Department of Molecular Biology and the Lewis-Sigler Institute of Integrative Genomics at Princeton. Home page at the Lewis-Sigler Institute Lab web page Princeton Profile Google Scholar publication page Twitter
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