The endotoxin, just by activating TLA4, so you're increasing nitric oxide, which is itself a free radical. You will directly start attacking a lot of the pufferin tissues and converting, generating all of these toxic aldehydes that we mentioned earlier. For chidroxine, one of them, but there are multiple others, which are now known to be a primary component of arterial plaque. Also, nitric oxide stimulates itself, tryptophilidroxylase. It stimulates something called NFKB, nuclear factor,. Kappa B, which is like the master regulator of the entire inflammatory cascade. And then what else? Oh, yeah, I wanted

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