The Role of Sedatives in TBI

Parex is a way of trying to look for a physiological proxy before you go for outcome. And the things that we have that look at from that study I showed you what have we shown to be associated with an improvement in Parex. It's mild, a mild hyperventilation. Hypothermia. High level sedation for ICP control but not a isometric. Not a herst suppressor and then vasopressa use for the CPP target. The thiocomar and other things like that don't seem to be associated. So whether or not they're related? We've never looked at that. That'd be a cool thing to do giving prop