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#14 - Robert Lustig, M.D., M.S.L.: fructose, processed food, NAFLD, and changing the food system

The Peter Attia Drive

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Metabolxandrome

There are some patients where that is the pathway. I actually think that's actually a rare way that this happens, maybe ten percent. We'll call it the atpagenic hypothesis of metabolxindro because clearly that patient is not insolent resistant at the atapocite. Yet, in fact, muscles don't need insolent to import glucos. If they did, then every diabetic would be paralyzed. The reason insolen works at the muscle is for import of meno acids, so for muscle growth, but not for muscle metabolism.

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