And looking at indemetrial

receptivity and the microbiom, is there a roll there for disbiosis and inflammatory changes in driving progestoron resistance? I mean, realize we're just at the tip of the iceberg on all this. In over the next few years, we're going to see so much more information coming through. But but what are your thoughts on the microbiome and the indemetria receptivity

side of things? That's a great question. And and the minutes not something i've spent a lot of time thinking about, couse, my space is not necessarily fertility driven time. But i do think that when we look at the endemetrium, and what we know on a microbium level, of both the endemetrial microbium and the vagina microbium is that they are connected in an up stream from the vagina to the endemetrium, and that they have common traits amongst them. And that we certainly have information in situations like indemetriosis, for example, where there are microbiam profiles of the endemetrium that are inflammatory and driven by microbes that are not conducive, as well to implantation and fertility. So within that, when we look at what we know about our indimaterial receptivity and projuced ouron resistancethat, there are these common markes of inflammatory changes and of tece reactions, and the suggestion that that sort of a resistance a lined with hal marks of somedmetriosis pictures as well. I think that the micro bom is definitely implicated, i just don't think we have the details of how exactly at this point. And thatth even the research on the enddometria microbam, talking about its roll ind fertilitis, is still in its infancy. But, i mean, it's certainly going to be an interesting space to watch. And as it comes to light, i i'm looking to be both excited and amazed.