Dose Dependent Seminiferous Tubula Atrophy and Reproductive Function

Fertility drug data is that at some dosage burden, actual destruction of seminiferous tubules occurs. In actual teicular functionactuallygets deteriorates. So i'm like, ok, is this happening with everything, including h c g? Because this is something that is obviously very comparable when we're comparing something that improves l h output and potentially even l h receptor expression in the testicles. Is h c g doing this too? Ecause this isSomething that, like the main go to fertility drug, that works very fucking well at what it does for intratisticular totastrum production. Like, what is that doing in rodente too? This was a study on no