Is It Better to Target TLR Nine on the Surface of Red Blood Cells to Modulate Inflammation?

Soi one of the things that they are arguing is, like, if now we know that this is a contributing fact, or could we target t lr nine on the surface of red blood cells to modulate inflammation? Because anti citiind antibodies, which they mention, have other consequences. So thar would be a less amunosuppressive way of down regulating inflammation. And this also sort of would get at the question that we talked about earlier, about is the point of thisno, the evolutionary point of this more to get rid of the selfre diane, or is it to have the red blood told bedana delivery device?