Now, I will say that it's not just straight up adverse effects that happen, but there were, there were some clear benefits. I mean, apart from the fact that my fasting glucose went down, it's also the case. It's also the case that my oxidative stress went down. And in the last episode, I explained that in a little bit more detail. I just didn't explain all the detail about the glucose tolerance test, but I had a, I had two organic acid tests before and after. And all the markers that I thought were indicating mild oxidative stress prior to the glucose tolerance testing disappeared at disappeared after the glucose tolerance testing. So I think that was in effect of the use of glucose in the pentasophosphate pathway and probably more significantly a positive effective insulin on glutathione synthesis and other other aspects of the antioxidant system. It also is looking last night and at my REM sleep. And my REM sleep is up significantly this year compared to last year. So last year's average was one hour and 32 minutes per night of REM sleep and this year's average is one hour and 45 minutes of REM sleep. But if I go to the month, then you know what, it's maybe this is due to wholly different reasons because my REM sleep bottomed out of the sleep. Bottom doubt in August. And then was kind of going up in a wavy pattern. Up to January where it peaked at one hour and 51 minutes, which was during my. Yeah, you know what, my REM sleep did peak. Let's look at the weeks it. It did peak in. It peaked at the end of January, just went straight up until the end of January. And so I guess the glucose tolerance testing does coincide with my REM sleep. So I'm not finishing going up to to a peak. It's still generally higher this year than last year, but I'm not sure what is the cause of that. But anyway, I do think the glucose tolerance testing decreased my oxidative stress. So there's that. So overall, the conclusions for my glucose tolerance, tolerance testing, my glucose testing clearly fails to support my hypothesis that biotin deficiency would cause glucose intolerance mitigated by per team. While protein might be having a small benefit for my glucose tolerance, it clearly is less than the benefit reported in the literature for healthy adults. However, if I try to integrate this with my review of the existing literature, the major issue seems to be whether the dose of protein is sufficient to match the dose of glucose. In the literature, it seems at least one gram of glucose is needed for every two grams of carbohydrate. Perhaps the bites in deficient state, the big difference would be that more protein than this is needed. That might explain why proteins seem defective in minimizing my glucose response to 30 grams of glucose right out of the gate, but was much less effective at neutralizing my response to 55 to 65 grams of glucose.