Hypokalemia

Thyrotoxic periodic paralysis is a function of hypokalemia. It develops through both direct and indirect impacts of thyroid hormone on the sodium-potassium ATPase channels, which leads to intracellular potassium shift. There's some metabolic factors like hyperinsulinemia and genetic factors like ion channel opathies that can further modify this risk. Ooh I'm sorry! That was absolutely amazing summary of such an interesting pathophysiology. And how do you balance out these electrolyte abnormalities induced by cellular shifts? On one end, it's really important to treat this,. But if you give too much, once all that potassium that's been shifted into the cells due to the